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- W2789190402 abstract "Preterm infants (i.e., born before <37 wk of gestation) are at increased risk of morbidity and mortality and long-term disabilities. Global prevalence of preterm birth (PTB) varies from 5 to 18 per cent. There are multiple aetiological causes and factors associated with PTB. Intrapartum infections are conventionally associated with PTB. However, maternal genotype modulates response to these infections. This review highlights the association of cytokine gene polymorphisms and their levels with PTB. Varying PTB rates across the different ethnic groups may be as a result of genetically mediated varying cytokines response to infections. Studies on genetic variations in tumour necrosis factor-alpha, interleukin-1 alpha (IL-1α), IL-1β, IL-6, IL-10 and toll-like receptor-4 genes and their association with PTB, have been reviewed. No single polymorphism of the studied genes was found to be associated with PTB. However, increased maternal levels of IL-1β and IL-6 and low levels of IL-10 have been found to be associated with PTB." @default.
- W2789190402 created "2018-03-06" @default.
- W2789190402 creator A5016646185 @default.
- W2789190402 creator A5044951490 @default.
- W2789190402 creator A5068479174 @default.
- W2789190402 date "2017-09-01" @default.
- W2789190402 modified "2023-10-05" @default.
- W2789190402 title "Interplay of cytokines in preterm birth." @default.
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- W2789190402 doi "https://doi.org/10.4103/ijmr.ijmr_1624_14" @default.
- W2789190402 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5793465" @default.
- W2789190402 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/29355137" @default.
- W2789190402 hasPublicationYear "2017" @default.
- W2789190402 type Work @default.