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• W2790053826 abstract "Acute promyelocytic leukemia is frequently associated with dizziness, fever, nausea, hematochezia and anemia. Blue light, or light with wavelengths of 400-480 nm, transmits high levels of energy. The aim of the present study was to determine the pro-apoptotic effects of blue light (wavelength, 456 nm; radiation power, 0.25 mW/cm2) and the underlying mechanisms in a human promyelocytic leukemia cell line (HL60). Blue light reduced the viability and enhanced the mortality of HL60 cells in a time-dependent manner. Exposure to blue light for 24 h caused depolarization of the mitochondrial membrane potential and the overproduction of reactive oxygen species in HL60 cells. In a nude mouse model, 9-day exposure to blue light markedly suppressed the growth of HL60-xenografted tumors; however, it had no effect on hepatic and renal tissues. In addition, blue light abrogated the expression of B-cell lymphoma (Bcl)-2 and Bcl extra-long, while enhancing the levels of Bcl-2-associated X protein, cytochrome c, and cleaved caspases-3 and -9 in tumor tissues. The results suggested that the pro-apoptotic effects of blue light in human promyelocytic leukemia cells may be associated with the mitochondrial apoptosis signaling pathway." @default.
• W2790053826 created "2018-03-29" @default.
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• W2790053826 date "2018-03-02" @default.
• W2790053826 modified "2023-09-28" @default.
• W2790053826 title "Blue light-induced apoptosis of human promyelocytic leukemia cells via the mitochondrial-mediated signaling pathway" @default.
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• W2790053826 doi "https://doi.org/10.3892/ol.2018.8162" @default.
• W2790053826 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5921239" @default.
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