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- W2793123503 abstract "In the event of an injury, normal tissues exit quiescent homeostasis and rapidly engage a complex stromal and immune program. These tissue repair responses are hijacked and become dysregulated in carcinogenesis to form a growth-supportive tumor microenvironment. In pancreatic ductal adenocarcinoma (PDA), which remains one of the deadliest major cancers, the microenvironment is a key driver of tumor maintenance that impedes many avenues of therapy. In this review, we outline recent efforts made to uncover the microenvironmental cross-talk mechanisms that support pancreatic cancer cells, and we detail the strategies that have been undertaken to help overcome these barriers." @default.
- W2793123503 created "2018-03-29" @default.
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- W2793123503 date "2018-07-01" @default.
- W2793123503 modified "2023-10-17" @default.
- W2793123503 title "Tumor cross-talk networks promote growth and support immune evasion in pancreatic cancer" @default.
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- W2793123503 doi "https://doi.org/10.1152/ajpgi.00416.2017" @default.
- W2793123503 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6109710" @default.
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