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- W2793562734 endingPage "24" @default.
- W2793562734 startingPage "1" @default.
- W2793562734 abstract "Glucose is the major energy substrate during embryogenesis and the embryo is dependent on glucose from the maternal circulation to ensure normal metabolism and growth. The placenta plays a key role in this nutrient transfer in mammals, both during embryogenesis and after the development of the chorio-allantoic placental circulation. Maternal hypoglycaemia is accompanied by foetal hypoglycaemia and maternal counter-regulatory measures including a priority to keep nutrients in the maternal circulation by restricting their transfer to the foetus. Concomitantly, the foetus initiates its own counter-regulatory attempt to secure nutrients for its development and survival. Despite these measures, there is a general decrease in nutrient transfer to the foetus, which may have severe consequences for foetal development such as malformations and delayed skeletal development." @default.
- W2793562734 created "2018-03-29" @default.
- W2793562734 creator A5010519662 @default.
- W2793562734 creator A5017050869 @default.
- W2793562734 creator A5035893451 @default.
- W2793562734 creator A5043589906 @default.
- W2793562734 date "2018-04-01" @default.
- W2793562734 modified "2023-09-27" @default.
- W2793562734 title "Effect of maternal hypoglycaemia during gestation on materno-foetal nutrient transfer and embryo-foetal development: Evidence from experimental studies focused primarily on the rat" @default.
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