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• W2793958408 abstract "FoxP3+ regulatory T cells (Tregs) are a subset of CD4+ T cells that can suppress proliferation and effector functions of T cells, B cells, NK cells, and antigen-presenting cells. Treg deficiency causes dramatic immunologic disease in both animal models and humans. As they are capable to suppress the function and the proliferation of conventional CD4+ and CD8+ T cells, Treg-based cell therapies are under evaluation for the treatment of various autoimmune diseases and are currently employed to prevent graft-versus-host disease (GvHD) in clinical trials of hematopoietic stem cell transplantation. Even though tumor necrosis factor-α (TNF-α) is well known for its pro-inflammatory role, recent studies show that it promotes Treg activation and suppressive function. In the present review, we discuss the role of TNF-α in Treg function and the possible implications on the actual treatments for immune-mediated diseases, with a particular attention to GvHD." @default.
• W2793958408 created "2018-03-29" @default.
• W2793958408 creator A5042367832 @default.
• W2793958408 creator A5043160139 @default.
• W2793958408 creator A5050917506 @default.
• W2793958408 creator A5070029054 @default.
• W2793958408 date "2018-02-28" @default.
• W2793958408 modified "2023-10-12" @default.
• W2793958408 title "The Effect of TNF-α on Regulatory T Cell Function in Graft-versus-Host Disease" @default.
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• W2793958408 doi "https://doi.org/10.3389/fimmu.2018.00356" @default.
• W2793958408 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5835761" @default.
• W2793958408 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/29541073" @default.
• W2793958408 hasPublicationYear "2018" @default.
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