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• W2799671608 abstract "This study investigated the inhibitory effects of kimchi bioactive compounds against endoplasmic reticulum (ER) stress-induced apoptosis in amyloid beta (Aβ)-injected mice. Mice received a single intracerebroventricular injection of Aβ25–35, except for the normal group. Mice were subjected to oral administration of 10 mg of capsaicin, 50 mg of 3-(4′-hydroxyl-3′,5′-dimethoxyphenyl)propionic acid (HDMPPA), 50 mg of quercetin, 50 mg of ascorbic acid, or 200 mg of kimchi methanol extract (KME) per kilogram of body weight for 2 weeks (n = 7 per group). In the in vitro blood–brain barrier (BBB) permeability test, all bioactive compounds penetrated the BBB except ascorbic acid. The protein expression level of APP, BACE, and p-Tau elevated by Aβ injection was decreased by kimchi bioactive compounds (P < 0.05). Quercetin, HDMPPA, and KME decreased oxidative stress, as indicated by ROS and TBARS levels (P < 0.05). The protein expression level of ER stress markers GRP78, p-PERK, p-eIF2α, XBP1, and CHOP and the proapoptotic molecules Bax, p-JNK, and cleaved caspases-3 and -9 decreased (P < 0.05). In contrast, the protein expression level of antiapoptotic molecules Bcl2 and cIAP increased (P < 0.05). These results were supported by histological analysis." @default.
• W2799671608 created "2018-05-17" @default.
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• W2799671608 date "2018-04-30" @default.
• W2799671608 modified "2023-10-17" @default.
• W2799671608 title "Bioactive Compounds of Kimchi Inhibit Apoptosis by Attenuating Endoplasmic Reticulum Stress in the Brain of Amyloid β-Injected Mice" @default.
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• W2799671608 doi "https://doi.org/10.1021/acs.jafc.8b01686" @default.
• W2799671608 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/29706080" @default.
• W2799671608 hasPublicationYear "2018" @default.
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