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• W280700028 abstract "Depletion of intracellular calcium stores such as the endoplasmic reticulum (ER) activates store-operated channels for Ca2+ entry across the plasma membrane (PM), as demonstrated in Fig. 8.1. This process is called the store-operated calcium entry (SOCE), a common and ubiquitous mechanism of regulating Ca2+ influx into cells [1, 7, 36, 38]. The best-studied store-operated channel (SOC) is the Ca2+ releaseactivated Ca2+ channel (CRAC) [5, 12, 13, 30, 38, 41, 53]. SOCE is a key feedback controller to stabilize ER Ca2+ and has been proposed as the main Ca2+ entry pathway in non-excitable cells [37, 38]. SOCE, originally known as capacitative calcium entry (CCE), was first proposed by Putney [42] and has been extensively studied later (for review, see Berridge [2, 3], Bird et al [4], Chakrabarti et al [6], Dirksen [9], Lewis [25], Parekh [37], Potier et al [39], Prakriya et al [40], Putney [43], and Shuttleworth et al [47])." @default.
• W280700028 created "2016-06-24" @default.
• W280700028 creator A5066152182 @default.
• W280700028 date "2012-01-01" @default.
• W280700028 modified "2023-09-27" @default.
• W280700028 title "Store-Operated Calcium Entry" @default.
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• W280700028 doi "https://doi.org/10.1007/978-88-470-2490-8_8" @default.
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