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- W2807478935 endingPage "267" @default.
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- W2807478935 abstract "Colorectal cancer (CRC) is the third most common cancer and the second leading cause of cancer-related death in the USA. It is of practical importance to identify novel therapeutic targets of CRC to develop new anti-cancer drugs and to discover novel biomarkers of CRC to develop new detection methods. Eicosanoids, which are metabolites of polyunsaturated fatty acids produced by cyclooxygenase (COX), lipoxygenase (LOX), and cytochrome P450 (CYP) enzymes, are important lipid-signaling molecules involved in the regulation of inflammation and tumorigenesis. Substantial studies have shown that the profiles of eicosanoids are deregulated in CRC, and the enzymes, metabolites, and receptors in the eicosanoid signaling cascade play critical roles in regulating colonic inflammation and colon tumorigenesis. In this review, we discuss the roles of the COX, LOX, and CYP pathways in the carcinogenesis of CRC." @default.
- W2807478935 created "2018-06-13" @default.
- W2807478935 creator A5011488688 @default.
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- W2807478935 creator A5031636414 @default.
- W2807478935 creator A5044229632 @default.
- W2807478935 creator A5073150091 @default.
- W2807478935 creator A5089510533 @default.
- W2807478935 date "2018-06-01" @default.
- W2807478935 modified "2023-10-12" @default.
- W2807478935 title "Eicosanoid signaling in carcinogenesis of colorectal cancer" @default.
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