Matches in SemOpenAlex for { <https://semopenalex.org/work/W2807552210> ?p ?o ?g. }
- W2807552210 abstract "Currently, the etiology of Alzheimer's disease (AD) is still elusive. Central insulin resistance has been determined to play an important role in the progress of AD. However, the mechanism underlying the development of disrupted insulin signaling pathways in AD is unclear. Suppressor of cytokine signaling 3 (SOCS3) is a member of the SOCS protein family that acts as a negative modulator of insulin signaling in sensitive tissues, such as hepatocytes and adipocytes. However, little is known about its role in neurological diseases. Recent evidence indicates that the level of SOCS3 is increased in the brains of individuals with AD, especially in areas with amyloid beta deposition, suggesting that SOCS3 may regulate the central insulin signaling pathways in AD. Here, we discuss the potential role of SOCS3 in AD and speculate that SOCS3 may be a promising therapeutic target for the treatment of AD." @default.
- W2807552210 created "2018-06-13" @default.
- W2807552210 creator A5019985136 @default.
- W2807552210 creator A5068456388 @default.
- W2807552210 creator A5076055381 @default.
- W2807552210 date "2018-06-20" @default.
- W2807552210 modified "2023-10-17" @default.
- W2807552210 title "Suppressor of Cytokine Signaling 3: Emerging Role Linking Central Insulin Resistance and Alzheimer’s Disease" @default.
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- W2807552210 doi "https://doi.org/10.3389/fnins.2018.00417" @default.
- W2807552210 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6020761" @default.
- W2807552210 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/29973864" @default.