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• W2808300529 abstract "Osteoporosis is a disease characterized by osteoclast-mediated low bone mass. The modulation of osteoclasts is important for the prevention or therapeutic treatment of loss of bone mass. Osteoclasts, which are bone-resorbing multinucleated cells, are differentiated from the hematopoietic stem cell monocyte/macrophage lineage by Receptor activator of nuclear factor kappa-B ligand (RANKL) expressed from osteoblasts and stromal cells. RANKL signaling ultimately activates nuclear factor of activated T Cells 1 (NFATc1), which is a master transcription factor in osteoclastogenesis. Betulin, a lupine type pentacyclic triterpenoid, was isolated from the bark of Betula platyphylla. Betulin inhibited RANKL-mediated osteoclast differentiation by downregulating NFATc1. Betulin may serve as a useful structural scaffold in the therapeutic agent development to prevention/treatment the osteoclast-mediated bone disorder." @default.
• W2808300529 created "2018-06-21" @default.
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• W2808300529 date "2018-06-15" @default.
• W2808300529 modified "2023-10-17" @default.
• W2808300529 title "Betulin Suppresses Osteoclast Formation via Down-Regulating NFATc1" @default.
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• W2808300529 doi "https://doi.org/10.3390/jcm7060154" @default.
• W2808300529 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6025465" @default.
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