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- W2808882009 abstract "Significance The identification of α7 nicotinic acetylcholine receptor (α7 NAChR) as the principal receptor for amyloid fibrils and small heat shock proteins clarifies their mechanism of immune suppression. The discovery of the receptor allows previous experiments using amyloid fibrils to be interpreted based on the extensive body of literature detailing the NAChR signaling pathway. In addition, the data further support the realization that amyloid fibrils are immune suppressive, which may be an important factor in neurodegenerative diseases. The demonstration that NAChR agonists are effective in limiting ischemia and autoimmunity emphasizes that the α7-specific partial agonists, which failed in clinical trials for Alzheimer’s disease and schizophrenia, could be reconsidered as therapeutics for a spectrum of inflammatory indications." @default.
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- W2808882009 date "2018-06-18" @default.
- W2808882009 modified "2023-09-27" @default.
- W2808882009 title "Identification of a common immune regulatory pathway induced by small heat shock proteins, amyloid fibrils, and nicotine" @default.
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- W2808882009 doi "https://doi.org/10.1073/pnas.1804599115" @default.
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