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- W2810242653 abstract "Contact urticaria syndrome, contact urticaria, and protein contact dermatitis are a heterogeneous group of immediate skin reactions clinically characterized by the presence of wheals, or eczema, or both, commonly limited to the exposed skin. Two pathogenic pathways have been described, which include a nonimmune-mediated mechanism and an immune-mediated mechanism. In the nonimmune-mediated mechanism, the agent directly causes skin reaction through vasogenic mediators, and therefore prior sensitization is not required to develop a skin reaction. Conversely, the immune-mediated mechanism consists of an IgE-mediated type I hypersensitivity reaction. This type of hypersensitivity requires an initial exposure to the antigen to induce a sensitization, causing the production of antigen-specific IgE that will induce mast cell degranulation after future exposures with the same antigen. Additionally, studies in vitro, as well as in vivo, have demonstrated supplementary roles of IgE beyond the immediate immune response subjected to the presence of a specific antigen. These antigen-independent roles of IgE have been shown to provide and prepare the perfect environment to induce the allergic response from the sensitization phase to amplification and perpetuation of this immune response. Herein, the roles of IgE in the immediate contact response, as well as those favoring its development, are reviewed." @default.
- W2810242653 created "2018-07-10" @default.
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- W2810242653 creator A5055779393 @default.
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- W2810242653 date "2018-01-01" @default.
- W2810242653 modified "2023-10-18" @default.
- W2810242653 title "Immunoglobulin E: Pathogenic Relevance in Immediate Contact Reactions" @default.
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- W2810242653 doi "https://doi.org/10.1007/978-3-319-89764-6_9" @default.
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