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- W2884952568 endingPage "762" @default.
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- W2884952568 abstract "Endometriosis is a chronic, inflammatory, estrogen-dependent disease characterized by the growth of endometrial tissue outside of the uterine cavity. Although the etiology of endometriosis remains elusive, immunological dysfunction has been proposed as a critical facilitator of ectopic lesion growth following retrograde menstruation of endometrial debris. However, it is not clear whether this immune dysfunction is a cause or consequence of endometriosis. Thus, here we provide in-depth insights into our current understanding of the immunopathophysiology of endometriosis and highlight challenges and opportunities for future research. With the explosion of successful immune-based therapies targeting various chronic inflammatory conditions, it is crucial to determine whether immune dysfunction can be therapeutically targeted in endometriosis." @default.
- W2884952568 created "2018-08-03" @default.
- W2884952568 creator A5005468871 @default.
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- W2884952568 creator A5062464806 @default.
- W2884952568 creator A5062775117 @default.
- W2884952568 creator A5080134110 @default.
- W2884952568 date "2018-09-01" @default.
- W2884952568 modified "2023-10-17" @default.
- W2884952568 title "The Immunopathophysiology of Endometriosis" @default.
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