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- W2885163402 abstract "Introduction: Patients with community-acquired pneumonia (CAP) are at increased risk of cardiovascular events; platelet activation has been suggested to play a role but the underlying mechanism is still unclear. Hypothesis: Our study hypothesis was that endotoxins (bacterial lipopolysaccharide, LPS) might play a role. Methods: Patients (n=100) with diagnosis of community-acquired pneumonia (CAP) through the emergency department (ED) and healty subjects (n=50) were consecutively recruited. Categorical variables were reported as counts (percentage) and continuous variables as means ± standard deviation (SD). The study was comprised 3 parts: 1) analysis of serum endotoxins in CAP patients versus controls; 2) ex vivo platelets aggregation in CAP patients and controls; 3) in vitro experiments to assess if LPS, in a range of concentrations detected in the blood of CAP patients, was capable of activating platelets. Results: CAP patients showed higher serum LPS and ex vivo platelet aggregation (PA) compared to co..." @default.
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- W2885163402 date "2015-11-10" @default.
- W2885163402 modified "2023-09-28" @default.
- W2885163402 title "Abstract 15209: Endotoxemia as Mechanism for Platelet Aggregation in Patients With Pneumonia" @default.
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