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- W2885180314 abstract "Background—Endothelial nitric oxide (NO)–mediated responses are impaired in arterioles of male spontaneously hypertensive rats (SHR), but they are still present in female SHR. We hypothesized that in vitro incubation of arterioles of male SHR with estrogen will restore NO-mediated responses by upregulation of endothelial NO synthase. Methods and Results—Responses to increases in perfusate flow (from 0 to 25 μL/min) and to the calcium ionophore A23187 (5×10−8 to 10−6 mol/L), norepinephrine (NE; 10−7 to 3×10−7 mol/L), sodium nitroprusside (SNP; 10−8 to 10−6 mol/L), and adenosine (ADO; 10−6 to 5×10−5 mol/L) were studied in cannulated and pressurized gracilis muscle arterioles (≈75 μm in diameter) isolated from 12-week-old male SHR before and after incubation with 10−9 mol/L 17β-estradiol (17β-E2) for 16 to 18 hours. After incubation with 17β-E2, basal diameter of arterioles was significantly increased (by ≈10%), and flow-induced dilation was significantly enhanced (79.8±2.9 versus 103.7±3.7 μm at 25 μL/min),..." @default.
- W2885180314 created "2018-08-22" @default.
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- W2885180314 date "2000-01-04" @default.
- W2885180314 modified "2023-09-23" @default.
- W2885180314 title "17β-Estradiol Restores Endothelial Nitric Oxide Release to Shear Stress in Arterioles of Male Hypertensive Rats" @default.
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