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- W2885514779 abstract "Background—The Rho/Rho-associated kinase (Rho-kinase) system is implicated in various cellular functions, including migration, proliferation, and apoptosis. Because a possible role of the system is suggested in neointima formation after vascular injury, we sought to examine whether a new specific Rho-kinase inhibitor, Y27632, prevents neointima formation of the balloon-injured rat carotid artery, and if so, to investigate the effects of Y27632 on migration, proliferation, and apoptosis of smooth muscle cells (SMCs) in the injured artery. Methods and Results—Y27632 was administered intraperitoneally from 1 day before to 14 days after vascular injury. Treatment with Y27632 inhibited phenylephrine-induced Rho-kinase activation in the carotid artery on the basis of immunoblotting against the phosphorylated myosin-binding subunit of myosin phosphatase. Y27632 markedly prevented neointima formation at days 7 and 14. In controls, BrdU+ proliferating and TUNEL+ apoptotic SMCs were transiently and coincidentally i..." @default.
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- W2885514779 date "2001-01-16" @default.
- W2885514779 modified "2023-09-23" @default.
- W2885514779 title "Role of Rho-Associated Kinase in Neointima Formation After Vascular Injury" @default.
- W2885514779 hasPublicationYear "2001" @default.
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