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• W2885874089 abstract "Background: Patients with systemic lupus erythematosus (SLE) are twice more likely to develop cardiovascular disease than the general population, even though their plasma LDL cholesterol (LDL-C) levels are usually not elevated. To delineate the mechanisms, we examined the chemical properties of their LDL. Methods and Results: LDL isolated from SLE patients (LDL-C, 105±33 mg/dL; n=24) exhibited greater mobility in agarose gel electrophoresis than LDL of healthy control subjects (LDL-C, 121±25 mg/dL; n=24), secondary to an increased distribution of L5 (2.30±1.3% vs. 0.7±0.3%; P<0.0001), the most electronegative subfraction of LDL identified by anion-exchange chromatography, in total LDL. CX3CL1 is a membrane-bound chemokine expressed in injured endothelial cells (ECs). CD16+ monocytes are CX3CR1-expressing cells that recognize CX3CL1. Compared with control, SLE patients had a twofold (P<0.001) increase in CX3CL1 and a threefold (P<0.0001) increase in CD16+ monocytes in the plasma. Moreover, there was a posi..." @default.
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• W2885874089 date "2014-11-25" @default.
• W2885874089 modified "2023-09-23" @default.
• W2885874089 title "Abstract 16069: Low-Density Lipoprotein of Systemic Lupus Erythematosus Patients Contains Lysophosphatidylcholine-Rich Lipid That Induces Overexpression of CX3CL1 in Endothelial Cells" @default.
• W2885874089 hasPublicationYear "2014" @default.
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