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- W2887126044 abstract "Gastroesophageal reflux impairs the mucosal barrier in the distal esophagus, allowing chronic exposure of the squamous epithelium to multitudinous stimulations and inducing chronic inflammation. Esophagitis is a response to inflammation of the esophageal squamous mucosa. Our study clarified that alcohol accumulation could aggravate the progress of esophagitis by inducing pyroptosis; however, Ac-YVAD-CMK, an inhibitor of caspase-1, could effectively suppress the expression of IL-1β and IL-18 both in vivo and in vitro, reducing the inflammatory response, which is promised to be an agent to inhibit the progression of esophagitis. Additionally, caspase-1-derived pyroptosis is involved in esophageal cancer." @default.
- W2887126044 created "2018-08-22" @default.
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- W2887126044 date "2018-01-01" @default.
- W2887126044 modified "2023-10-17" @default.
- W2887126044 title "Alcohol accumulation promotes esophagitis via pyroptosis activation" @default.
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- W2887126044 doi "https://doi.org/10.7150/ijbs.24347" @default.
- W2887126044 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6097477" @default.
- W2887126044 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/30123073" @default.
- W2887126044 hasPublicationYear "2018" @default.
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