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- W2887388617 endingPage "980" @default.
- W2887388617 startingPage "969" @default.
- W2887388617 abstract "Skeletal muscle fitness and plasticity is an important determinant of human health and disease. Mitochondria are essential for maintaining skeletal muscle energy homeostasis by adaptive re-programming to meet the demands imposed by a myriad of physiologic or pathophysiological stresses. Skeletal muscle mitochondrial dysfunction has been implicated in the pathogenesis of many diseases, including muscular dystrophy, atrophy, type 2 diabetes, and aging-related sarcopenia. Notably, exercise counteracts the effects of many chronic diseases on skeletal muscle mitochondrial function. Recent studies have revealed a finely tuned regulatory network that orchestrates skeletal muscle mitochondrial biogenesis and function in response to exercise and in disease states. In addition, increasing evidence suggests that mitochondria also serve to “communicate” with the nucleus and mediate adaptive genomic re-programming. Here we review the current state of knowledge relevant to the dynamic remodeling of skeletal muscle mitochondria in response to exercise and in disease states." @default.
- W2887388617 created "2018-08-22" @default.
- W2887388617 creator A5000014440 @default.
- W2887388617 creator A5028299159 @default.
- W2887388617 creator A5059667799 @default.
- W2887388617 creator A5066943426 @default.
- W2887388617 date "2018-08-14" @default.
- W2887388617 modified "2023-10-18" @default.
- W2887388617 title "Skeletal muscle mitochondrial remodeling in exercise and diseases" @default.
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