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- W2889807316 abstract "Ethanol exerts biological effects through multiple receptors, including ion channels. Alcohol intoxication may induce atrial fibrillation prone cellular electrophysiological changes and arrhythmias, primarily AF. Modifications of inward rectifier potassium currents have been implicated in the pathogenesis of AF. However, the mechanisms underlying the potential association between acute ethanol exposure and AF remain unclear. Therefore, we hypothesized that alcohol may affect acetylcholine-activated K+ current in atrial cardiomyocytes. Methods: IKACh current was recorded using a nystatin-perforated whole cell patch-clamp technique following activation by ACh. After the measurement of baseline IKACh current, cardiomyocytes were exposed to ethanol (100 mM for 10 min) and IKACh current was re-measured. Current-voltage (I-V) curves were obtained from the current response induced by voltage ramps between –120 and +60 mV from the holding potential of –40 mV. Results: Application of ACh (10 μM) to the bath soluti..." @default.
- W2889807316 created "2018-09-27" @default.
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- W2889807316 date "2016-11-11" @default.
- W2889807316 modified "2023-09-24" @default.
- W2889807316 title "Abstract 18376: Ethanol Induces Prolongation of the Plateau Phase of Acetylcholine-Activated K+ Current in Mouse Atrial Cardiomyocytes" @default.
- W2889807316 hasPublicationYear "2016" @default.
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