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- W2890150508 abstract "Herpes simplex virus 1 (HSV-1) is among the most common human pathogens. Little is known regarding the exact mechanism by which this virus evades the human immune system, particularly the innate immune system. We reported previously that HSV-1 employs its protein kinase US3 to modulate the expression of the key antigen-presenting molecule, CD1d, so as to evade the antiviral function of NKT cells. Here we demonstrated that the virus has coevolved with the human CD1d and NKT cell system and that NKT cells indeed play potent roles in anti-HSV immune responses. These studies point to the great potential of exploring NKT cell ligands as adjuvants for HSV vaccines." @default.
- W2890150508 created "2018-09-27" @default.
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- W2890150508 date "2018-11-15" @default.
- W2890150508 modified "2023-10-01" @default.
- W2890150508 title "Herpes Simplex Virus 1 Specifically Targets Human CD1d Antigen Presentation To Enhance Its Pathogenicity" @default.
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- W2890150508 doi "https://doi.org/10.1128/jvi.01490-18" @default.
- W2890150508 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6206489" @default.
- W2890150508 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/30185591" @default.
- W2890150508 hasPublicationYear "2018" @default.