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- W2890736750 abstract "Abstract Human immunodeficiency virus (HIV) infection is characterized by dysfunctional B cell responses. Here we show that chronic simian immunodeficiency virus (SIV) infection is characterized by an expansion of either lymph node germinal center (GC) B cells that co‐express Bcl6, Ki‐67 and IL‐21R and correlate with expanded T follicular helper (Tfh) cells or B cells that lack Bcl6, Ki‐67 and IL‐21R but express high levels of anti‐apoptotic Bcl2 that negatively correlate with Tfh cells. The lack of Tfh cells likely contributes to persistence of dysfunctional non‐proliferating B cells during chronic infection. These findings have implications for protective immunity in HIV‐infected individuals who harbour low frequencies of Tfh cells." @default.
- W2890736750 created "2018-09-27" @default.
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- W2890736750 date "2018-09-06" @default.
- W2890736750 modified "2023-09-26" @default.
- W2890736750 title "Chronic simian immunodeficiency virus infection is associated with contrasting phenotypes of dysfunctional Bcl6<sup>+</sup> germinal center B cells or Bcl6<sup>−</sup>Bcl2<sup>+</sup> non‐germinal center B cells" @default.
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- W2890736750 doi "https://doi.org/10.1111/jcmm.13844" @default.
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