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- W2891031763 abstract "Abstract Cell plasticity—the ability to reversibly assume different cellular phenotypes—has widely been studied in the context of development, wound repair, and cancer metastasis. Often this plasticity is exhibited as an epithelial-to-mesenchymal transition (EMT), where cells with an epithelial phenotype transition to display a mesenchymal phenotype while maintaining the capacity to reassume their epithelial state. This transition between cellular phenotypes is often regulated by environmental cues triggered during stress, which promote a mesenchymal state to aid in cell survival and repair of a tissue. In this chapter, we explore phenotypic plasticity and its consequences in the epithelium of the ovary and the fallopian tube during ovulation and ovulatory wound repair. We then discuss cellular plasticity in the context of ovarian cancer, how it impacts cancer progression, and how this affects the design and efficacy of ovarian cancer therapies." @default.
- W2891031763 created "2018-09-27" @default.
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- W2891031763 date "2019-01-01" @default.
- W2891031763 modified "2023-09-24" @default.
- W2891031763 title "Phenotypic Plasticity and the Origins and Progression of Ovarian Cancer" @default.
- W2891031763 doi "https://doi.org/10.1016/b978-0-12-813209-8.00033-9" @default.
- W2891031763 hasPublicationYear "2019" @default.
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