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• W2891165745 endingPage "3352" @default.
• W2891165745 startingPage "3339" @default.
• W2891165745 abstract "Abstract The competitive endogenous RNA (ceRNA) hypothesis suggests that a long noncoding RNA (lncRNA) can function as sinks for pools of microRNAs (miRNAs); thereby, in the presence of ceRNA, messenger RNAs (mRNAs) targeted by specific miRNAs can liberate and translate to protein. Maternally expressed gene 3 ( MEG3 ) is a lncRNA, which its expression has been detected in various normal tissues, while it is lost or downregulated in human tumors. The MEG3 is an imprinted gene which, is methylated and suppressed by DNA methyltransferases (DNMTs) family. Also, miRNAs are involved in the regulation of MEG3 gene expression. Interestingly, the lncRNA MEG3 (lnc‐MEG3), as a ceRNA affects various cell processes such as proliferation, apoptosis, and angiogenesis by sponging miRNAs. These miRNAs, in turn, regulate different mRNAs in different pathways. This review focuses on the interaction between lnc‐MEG3 and experimentally validated miRNAs. In addition, the discussion supplemented by some data obtained from mirPath (v.3) and TarBase (v.8) databanks to provide more details about the pathways affected by this ceRNA." @default.
• W2891165745 created "2018-09-27" @default.
• W2891165745 creator A5012898980 @default.
• W2891165745 creator A5028853982 @default.
• W2891165745 creator A5039351806 @default.
• W2891165745 date "2018-09-19" @default.
• W2891165745 modified "2023-10-14" @default.
• W2891165745 title "Interaction of long noncoding RNA MEG3 with miRNAs: A reciprocal regulation" @default.
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• W2891165745 doi "https://doi.org/10.1002/jcb.27604" @default.
• W2891165745 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/30230601" @default.
• W2891165745 hasPublicationYear "2018" @default.
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