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• W2891792826 startingPage "1036" @default.
• W2891792826 abstract "Boron is a micronutrient that is required for the normal growth and development of vascular plants, but its precise functions remain a subject of debate. One established role for boron is in the cell wall where it forms a diester cross-link between two monomers of the low-abundance pectic polysaccharide rhamnogalacturonan-II (RG-II). The inability of RG-II to properly assemble into a dimer results in the formation of cell walls with abnormal biochemical and biomechanical properties and has a severe impact on plant productivity. Here we describe the effects on RG-II structure and cross-linking and on the growth of plants in which the expression of a GDP-sugar transporter (GONST3/GGLT1) has been reduced. In the GGLT1-silenced plants the amount of L-galactose in side-chain A of RG-II is reduced by up to 50%. This leads to a reduction in the extent of RG-II cross-linking in the cell walls as well as a reduction in the stability of the dimer in the presence of calcium chelators. The silenced plants have a dwarf phenotype, which is rescued by growth in the presence of increased amounts of boric acid. Similar to the mur1 mutant, which also disrupts RG-II cross-linking, GGLT1-silenced plants display a loss of cell wall integrity under salt stress. We conclude that GGLT1 is probably the primary Golgi GDP-L-galactose transporter, and provides GDP-L-galactose for RG-II biosynthesis. We propose that the L-galactose residue is critical for RG-II dimerization and for the stability of the borate cross-link." @default.
• W2891792826 created "2018-09-27" @default.
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• W2891792826 date "2018-10-20" @default.
• W2891792826 modified "2023-10-02" @default.
• W2891792826 title "Suppression of Arabidopsis GGLT1 affects growth by reducing the L-galactose content and borate cross-linking of rhamnogalacturonan-II" @default.
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• W2891792826 doi "https://doi.org/10.1111/tpj.14088" @default.
• W2891792826 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6263843" @default.
• W2891792826 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/30203879" @default.
• W2891792826 hasPublicationYear "2018" @default.
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