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- W2891928151 endingPage "99" @default.
- W2891928151 startingPage "89" @default.
- W2891928151 abstract "Renal cell carcinoma (RCC) is highly dependent on angiogenesis, due to the overactivation of the VHL/HIF/VEGF/VEGFRs axis; this justifies the marked sensitivity of this neoplasm to antiangiogenic agents which, however, ultimately fail to control tumor growth. RCC also frequently shows alterations in the mTOR signaling pathway, and mTOR inhibitors have shown a similar pattern of initial activity/late failure as pure antiangiogenic agents. Understanding mechanisms of resistance to these agents would be key to improve the outcome of our patients. Circulating endothelial cells are a family of mainly bone marrow-derived progenitors, which have been postulated to be responsible of the reactivation of angiogenesis in different tumors. In this review, we shall discuss the complex nature and function of these cells, the evidence pro and contra their contribution to tumor vascularization, especially as far as RCC is concerned, and their possible role in determining resistance to presently available treatments." @default.
- W2891928151 created "2018-09-27" @default.
- W2891928151 creator A5018910230 @default.
- W2891928151 creator A5019241495 @default.
- W2891928151 creator A5026120374 @default.
- W2891928151 creator A5028393977 @default.
- W2891928151 creator A5032741879 @default.
- W2891928151 creator A5065426803 @default.
- W2891928151 date "2018-12-01" @default.
- W2891928151 modified "2023-10-16" @default.
- W2891928151 title "The role of endothelial colony forming cells in kidney cancer’s pathogenesis, and in resistance to anti-VEGFR agents and mTOR inhibitors: A speculative review" @default.
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