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- W2892306013 endingPage "2865" @default.
- W2892306013 startingPage "2865" @default.
- W2892306013 abstract "In cystic fibrosis (CF), impaired airway surface hydration (ASL) and mucociliary clearance that promote chronic bacterial colonization, persistent inflammation, and progressive structural damage to the airway wall architecture are typically explained by ion transport abnormalities related to the mutation of the gene coding for the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) channel. However, the progressive and unrelenting inflammation of the CF airway begins early in life, becomes persistent, and is excessive relative to the bacterial burden. Intrinsic abnormalities of the inflammatory response in cystic fibrosis have been suggested but the mechanisms involved remain poorly understood. This review aims to give an overview of the recent advances in the understanding of the defective resolution of inflammation in CF including the abnormal production of specialized pro-resolving lipid mediators (lipoxin and resolvin) and their impact on the pathogenesis of the CF airway disease." @default.
- W2892306013 created "2018-09-27" @default.
- W2892306013 creator A5026101627 @default.
- W2892306013 creator A5057176464 @default.
- W2892306013 date "2018-09-21" @default.
- W2892306013 modified "2023-09-26" @default.
- W2892306013 title "Specialized Pro-Resolving Lipid Mediators in Cystic Fibrosis" @default.
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- W2892306013 doi "https://doi.org/10.3390/ijms19102865" @default.
- W2892306013 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6213393" @default.
- W2892306013 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/30241412" @default.
- W2892306013 hasPublicationYear "2018" @default.
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