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- W2892955102 abstract "It is well known that animal viruses enter host cells via endocytosis, whereas plant viruses require physical assistance, such as human and insect activities, to penetrate the host cell to establish their infection. In this study, we report that the endocytosis pathway is also involved in virus infection in plants. We show that plant potyviruses recruit endocytosis dynamin-like proteins to support their infection. Depletion of them by knockout of the corresponding genes suppresses virus replication, whereas overexpression of them enhances virus replication and intercellular movement. We also demonstrate that the dynamin-like proteins interact with several viral proteins that are essential for virus replication and cell-to-cell movement. We further show that treatment of a dynamin-specific inhibitor disrupts endocytosis and inhibits virus replication and intercellular movement. Therefore, the dynamin-like proteins are novel host factors of potyviruses. The corresponding genes may be manipulated using advanced biotechnology to control potyviral diseases." @default.
- W2892955102 created "2018-10-05" @default.
- W2892955102 creator A5006016361 @default.
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- W2892955102 date "2018-12-01" @default.
- W2892955102 modified "2023-10-16" @default.
- W2892955102 title "Dynamin-Like Proteins of Endocytosis in Plants Are Coopted by Potyviruses To Enhance Virus Infection" @default.
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- W2892955102 doi "https://doi.org/10.1128/jvi.01320-18" @default.
- W2892955102 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6232491" @default.
- W2892955102 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/30258010" @default.
- W2892955102 hasPublicationYear "2018" @default.
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