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• W2893156674 abstract "The mechanism by which c-Myc exerts its oncogenic functions is not completely clear and different hypotheses are still under investigation. The knowledge of the capacity of c-Myc to bind exclusively E-box sequences determined the discrepancy between, on the one hand, genomic studies showing the binding of c-Myc to all active promoters and, on the other hand, the evidence that only 60% or less of the binding sites have E-box sequences.In this review, we provide support to the hypothesis that the cooperation of c-Myc with transcriptional cofactors mediates c-Myc-induced cellular functions. We produce evidence that recently identified cofactors are involved in c-Myc control of survival mechanisms of cancer cells.The identification of new c-Myc cofactors could favor the development of therapeutic strategies able to compensate the difficulty of targeting c-Myc." @default.
• W2893156674 created "2018-10-05" @default.
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• W2893156674 date "2018-09-27" @default.
• W2893156674 modified "2023-10-14" @default.
• W2893156674 title "Recent advances in searching c-Myc transcriptional cofactors during tumorigenesis" @default.
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• W2893156674 doi "https://doi.org/10.1186/s13046-018-0912-2" @default.
• W2893156674 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6161371" @default.
• W2893156674 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/30261904" @default.
• W2893156674 hasPublicationYear "2018" @default.
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