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- W2894965090 endingPage "79" @default.
- W2894965090 startingPage "71" @default.
- W2894965090 abstract "Virus infection contributes to nearly 15% of human cancers worldwide. Many of the oncogenic viruses tend to cause cancer in immunosuppressed individuals, but maintain asymptomatic, persistent infection for decades in the general population. In this review, we discuss the tactics employed by two small DNA tumor viruses, Human papillomavirus (HPV) and Merkel cell polyomavirus (MCPyV), to establish persistent infection. We will also highlight recent key findings as well as outstanding questions regarding the mechanisms by which HPV and MCPyV evade host immune control to promote their survival. Since persistent infection enables virus-induced tumorigenesis, identifying the mechanisms by which small DNA tumor viruses achieve latent infection may inform new approaches for preventing and treating their respective human cancers." @default.
- W2894965090 created "2018-10-12" @default.
- W2894965090 creator A5002397034 @default.
- W2894965090 creator A5058310813 @default.
- W2894965090 creator A5071037763 @default.
- W2894965090 date "2018-10-01" @default.
- W2894965090 modified "2023-10-14" @default.
- W2894965090 title "Mechanisms of persistence by small DNA tumor viruses" @default.
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