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- W2895965610 endingPage "40" @default.
- W2895965610 startingPage "25" @default.
- W2895965610 abstract "Compared to the tidy and immunologically silent death during apoptosis, necrosis seems like a chaotic and unorganized demise. However, we now recognize that there is a method to its madness, as many forms of necrotic cell death are indeed programmed and function beyond lytic cell death to support homeostasis and immunity. Inherently more immunogenic than their apoptotic counterpart, programmed necrosis, such as necroptosis, pyroptosis, ferroptosis, and NETosis, releases inflammatory cytokines and danger-associated molecular patterns (DAMPs), skewing the milieu to a pro-inflammatory state. Moreover, impaired clearance of dead cells often leads to inflammation. Importantly, these pathways have all been implicated in inflammatory and autoimmune diseases, therefore careful understanding of their molecular mechanisms can have long lasting effects on how we interpret their role in disease and how we translate these mechanisms into therapy." @default.
- W2895965610 created "2018-10-26" @default.
- W2895965610 creator A5067443290 @default.
- W2895965610 creator A5071157110 @default.
- W2895965610 creator A5090651281 @default.
- W2895965610 date "2018-10-22" @default.
- W2895965610 modified "2023-10-16" @default.
- W2895965610 title "Programmed Necrosis and Disease:We interrupt your regular programming to bring you necroinflammation" @default.
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