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- W2896119560 endingPage "2197" @default.
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- W2896119560 abstract "Atherosclerosis is characterized by the retention of modified lipoproteins in the arterial wall. These modified lipoproteins activate resident macrophages and the recruitment of monocyte-derived cells, which differentiate into mononuclear phagocytes that ingest the deposited lipoproteins to become “foam cells”: a hallmark of this disease. In this Part 2 of a 4-part review series covering the macrophage in cardiovascular disease, we critically review the contributions and relevant pathobiology of monocytes, macrophages, and foam cells as relevant to atherosclerosis. We also review evidence that via various pathways, a failure of the resolution of inflammation is an additional key aspect of this disease process. Finally, we consider the likely role played by genomics and biological networks in controlling the macrophage phenotype in atherosclerosis. Collectively, these data provide substantial insights on the atherosclerotic process, while concurrently offering numerous molecular and genomic candidates that appear to hold great promise for selective targeting as clinical therapies." @default.
- W2896119560 created "2018-10-26" @default.
- W2896119560 creator A5035258831 @default.
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- W2896119560 creator A5088448170 @default.
- W2896119560 creator A5090194847 @default.
- W2896119560 date "2018-10-01" @default.
- W2896119560 modified "2023-10-11" @default.
- W2896119560 title "Macrophage Trafficking, Inflammatory Resolution, and Genomics in Atherosclerosis" @default.
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