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- W2896335144 abstract "Renal segmental metabolism is reflected by the complex distribution of the main energy pathways along the nephron, with fatty acid oxidation preferentially used in the cortex area. Ischemia/reperfusion injury (IRI) is due to the restriction of renal blood flow, rapidly leading to a metabolic switch toward anaerobic conditions. Subsequent unbalance between energy demand and oxygen/nutrient delivery compromises kidney cell functions, resulting in a complex inflammatory cascade including the production of reactive oxygen species (ROS). Renal IRI especially involves lipid accumulation. Lipid peroxidation is one of the major events of ROS-associated tissue injury. Here, we briefly review the current knowledge of renal cell lipid metabolism in normal and ischemic conditions. Next, we focus on renal lipid-associated injury, with emphasis on its mechanisms and consequences during the course of IRI. Finally, we discuss preclinical observations aiming at preventing and/or attenuating lipid-associated IRI." @default.
- W2896335144 created "2018-10-26" @default.
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- W2896335144 date "2018-12-01" @default.
- W2896335144 modified "2023-10-15" @default.
- W2896335144 title "What we need to know about lipid-associated injury in case of renal ischemia-reperfusion" @default.
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- W2896335144 doi "https://doi.org/10.1152/ajprenal.00322.2018" @default.
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