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- W2896689039 endingPage "944" @default.
- W2896689039 startingPage "931" @default.
- W2896689039 abstract "Alzheimer’s disease (AD), the most common form of dementia, is characterized by extracellular β-amyloid plaques and intracellular neurofibrillary tangles (NFTs), which are considered as major targets for AD therapies. However, no effective therapy is available to cure or prevent the progression of AD up until now. Accumulation of NFTs, which consist of abnormally hyperphosphorylated tau, is directly correlated with the degree of dementia in AD patients. Emerging evidence indicates that the prion-like seeding and spreading of tau pathology may be the key driver of AD. In the past decades, greater understanding of tau pathway reveals new targets for the development of specific therapies. Here, we review the recent research progress in the mechanism underlying tau pathology in AD and briefly introduce tau-based therapeutics." @default.
- W2896689039 created "2018-10-26" @default.
- W2896689039 creator A5018804020 @default.
- W2896689039 creator A5057271155 @default.
- W2896689039 date "2018-10-11" @default.
- W2896689039 modified "2023-10-16" @default.
- W2896689039 title "Pathological Changes of Tau Related to Alzheimer’s Disease" @default.
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