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- W289676507 abstract "Prostaglandin E 2 and prostacyclin are inflammatory mediators and injected into the rat paw induce hyperalgesia. Because prostaglandin hyperalgesia in our model (rat paw pressure test) develops slowly, it is likely that the agonist has already been removed from the biophase at the time that the hyperalgesia response reaches the plateau (1–3 hours). The hyperalgesia induced by prostaglandins is thought to be due to increased levels of cAMP/Ca 2+ at the nociceptors. Treatment with cycloheximide blocks the hyperalgesia effects of prostaglandins but not that induced by Db-cAMP. These results are consistent with the suggestion that prostaglandin hyperalgesia is dependent on the triggering of some metabolic process leading to the synthesis of some unkown neuronal factor which enhances the sensitivity of the pain receptors to mechanical or chemical stimulation." @default.
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- W289676507 date "1982-01-01" @default.
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- W289676507 title "Pain and Inflammation: a New Concept" @default.
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- W289676507 doi "https://doi.org/10.1016/b978-0-08-028024-0.50017-7" @default.
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