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- W2897100360 abstract "Blood vessel epicardial substance (BVES) is a tight-junction associated protein that was originally discovered from a cDNA screen of the developing heart. Research over the last decade has shown that not only is BVES is expressed in cardiac and skeletal tissue, but BVES is also is expressed throughout the gastrointestinal epithelium. Mice lacking BVES sustain worse intestinal injury and inflammation. Furthermore, BVES is suppressed in gastrointestinal cancers, and mouse modeling has shown that loss of BVES promotes tumor formation. Recent work from multiple laboratories has revealed that BVES can regulate several molecular pathways, including cAMP, WNT, and promoting the degradation of the oncogene, c-Myc. This review will summarize our current understanding of how BVES regulates the intestinal epithelium and discuss how BVES functions at the molecular level to preserve epithelial phenotypes and suppress tumorigenesis." @default.
- W2897100360 created "2018-10-26" @default.
- W2897100360 creator A5032861209 @default.
- W2897100360 creator A5037091749 @default.
- W2897100360 creator A5076258077 @default.
- W2897100360 date "2018-10-02" @default.
- W2897100360 modified "2023-09-27" @default.
- W2897100360 title "Blood Vessel Epicardial Substance (BVES) in junctional signaling and cancer" @default.
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- W2897100360 doi "https://doi.org/10.1080/21688370.2018.1499843" @default.
- W2897100360 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6389126" @default.
- W2897100360 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/30307367" @default.
- W2897100360 hasPublicationYear "2018" @default.
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