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• W2897613550 abstract "The cornea is an extraordinary component of vision that functions as the principal barrier to pathogens in the eye while allowing light transmission into the retina. Understanding the cellular and molecular mechanisms that maintain homeostasis in this tissue is the subject of intense scientific study given the high prevalence of corneal disease. Over the past decade, the interactions between lectins and glycans on plasma membranes have emerged as important regulatory factors in corneal biology. In particular, members of the galectin family have been shown to bind multiple β-galactoside-containing receptors to regulate immunopathological processes associated with viral and bacterial infection, transplantation, wound healing, dry eye, angiogenesis, and lymphangiogenesis. In this review, we describe the current understanding of how these surface interactions intersect with different pathways to activate unique cellular responses in cornea as well as their potential therapeutic implications." @default.
• W2897613550 created "2018-10-26" @default.
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• W2897613550 creator A5058971301 @default.
• W2897613550 date "2018-10-08" @default.
• W2897613550 modified "2023-09-25" @default.
• W2897613550 title "Lectin-Glycan Interactions in Corneal Infection and Inflammation" @default.
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• W2897613550 doi "https://doi.org/10.3389/fimmu.2018.02338" @default.
• W2897613550 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6186829" @default.
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• W2897613550 hasPublicationYear "2018" @default.
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