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- W2897775584 abstract "Activated by AMP-dependent and -independent mechanisms, AMP-activated protein kinase (AMPK) plays a central role in the regulation of cellular bioenergetics and cellular survival. AMPK regulates a diverse set of signaling networks that converge to epigenetically mediate transcriptional events. Reversible histone and DNA modifications, such as acetylation and methylation, result in structural chromatin alterations that influence transcriptional machinery access to genomic regulatory elements. The orchestration of these epigenetic events differentiates physiological from pathophysiological phenotypes. AMPK phosphorylation of histones, DNA methyltransferases and histone post-translational modifiers establish AMPK as a key player in epigenetic regulation. This review focuses on the role of AMPK as a mediator of cellular survival through its regulation of chromatin remodeling and the implications this has for health and disease." @default.
- W2897775584 created "2018-10-26" @default.
- W2897775584 creator A5016570069 @default.
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- W2897775584 creator A5076955147 @default.
- W2897775584 creator A5082132858 @default.
- W2897775584 date "2018-10-19" @default.
- W2897775584 modified "2023-10-12" @default.
- W2897775584 title "AMPK: An Epigenetic Landscape Modulator" @default.
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- W2897775584 doi "https://doi.org/10.3390/ijms19103238" @default.
- W2897775584 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6214086" @default.
- W2897775584 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/30347687" @default.
- W2897775584 hasPublicationYear "2018" @default.
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