FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W2897974085> ?p ?o ?g. }

• W2897974085 endingPage "3227" @default.
• W2897974085 startingPage "3227" @default.
• W2897974085 abstract "Recently, gaseous signaling molecules, such as carbon monoxide (CO), nitric oxide (NO), and hydrogen sulfide (H2S), which were previously considered to be highly toxic, have been of increasing interest due to their beneficial effects at low concentrations. These so-called gasotransmitters affect many cellular processes, such as apoptosis, proliferation, cytoprotection, oxygen sensing, ATP synthesis, and cellular respiration. It is thought that mitochondria, specifically their respiratory complexes, constitute an important target for these gases. On the other hand, increasing evidence of a cytoprotective role for mitochondrial potassium channels provides motivation for the analysis of the role of gasotransmitters in the regulation of channel function. A number of potassium channels have been shown to exhibit activity within the inner mitochondrial membrane, including ATP-sensitive potassium channels, Ca2+-activated potassium channels, voltage-gated Kv potassium channels, and TWIK-related acid-sensitive K+ channel 3 (TASK-3). The effects of these channels include the regulation of mitochondrial respiration and membrane potential. Additionally, they may modulate the synthesis of reactive oxygen species within mitochondria. The opening of mitochondrial potassium channels is believed to induce cytoprotection, while channel inhibition may facilitate cell death. The molecular mechanisms underlying the action of gasotransmitters are complex. In this review, we focus on the molecular mechanisms underlying the action of H2S, NO, and CO on potassium channels present within mitochondria." @default.
• W2897974085 created "2018-10-26" @default.
• W2897974085 creator A5008833361 @default.
• W2897974085 creator A5067612021 @default.
• W2897974085 creator A5091114714 @default.
• W2897974085 date "2018-10-18" @default.
• W2897974085 modified "2023-10-04" @default.
• W2897974085 title "Gas Signaling Molecules and Mitochondrial Potassium Channels" @default.
• W2897974085 cites W1195621313 @default.
• W2897974085 cites W1501532586 @default.
• W2897974085 cites W1546398187 @default.
• W2897974085 cites W1560088953 @default.
• W2897974085 cites W1639174764 @default.
• W2897974085 cites W1655162787 @default.
• W2897974085 cites W1883147193 @default.
• W2897974085 cites W1921020717 @default.
• W2897974085 cites W1961727917 @default.
• W2897974085 cites W1964320031 @default.
• W2897974085 cites W1964722626 @default.
• W2897974085 cites W1965100677 @default.
• W2897974085 cites W1969257702 @default.
• W2897974085 cites W1971770866 @default.
• W2897974085 cites W1973927461 @default.
• W2897974085 cites W1974210120 @default.
• W2897974085 cites W1974361621 @default.
• W2897974085 cites W1975424513 @default.
• W2897974085 cites W1976654748 @default.
• W2897974085 cites W1977328014 @default.
• W2897974085 cites W1980987756 @default.
• W2897974085 cites W1981860537 @default.
• W2897974085 cites W1981886269 @default.
• W2897974085 cites W1982895791 @default.
• W2897974085 cites W1986391901 @default.
• W2897974085 cites W1986492446 @default.
• W2897974085 cites W1987095470 @default.
• W2897974085 cites W1987188025 @default.
• W2897974085 cites W1987777210 @default.
• W2897974085 cites W1990606266 @default.
• W2897974085 cites W1991375427 @default.
• W2897974085 cites W1992255196 @default.
• W2897974085 cites W1994968070 @default.
• W2897974085 cites W1997023449 @default.
• W2897974085 cites W1999675865 @default.
• W2897974085 cites W2000588392 @default.
• W2897974085 cites W2000860669 @default.
• W2897974085 cites W2001277810 @default.
• W2897974085 cites W2001959488 @default.
• W2897974085 cites W2003282272 @default.
• W2897974085 cites W2005904261 @default.
• W2897974085 cites W2006754144 @default.
• W2897974085 cites W2009065530 @default.
• W2897974085 cites W2011279942 @default.
• W2897974085 cites W2011481428 @default.
• W2897974085 cites W2018205667 @default.
• W2897974085 cites W2019176942 @default.
• W2897974085 cites W2020309430 @default.
• W2897974085 cites W2020444237 @default.
• W2897974085 cites W2021371950 @default.
• W2897974085 cites W2024104987 @default.
• W2897974085 cites W2025368509 @default.
• W2897974085 cites W2025567913 @default.
• W2897974085 cites W2026945507 @default.
• W2897974085 cites W2028095836 @default.
• W2897974085 cites W2030344907 @default.
• W2897974085 cites W2031822790 @default.
• W2897974085 cites W2036033897 @default.
• W2897974085 cites W2041553103 @default.
• W2897974085 cites W2043100807 @default.
• W2897974085 cites W2049251974 @default.
• W2897974085 cites W2049923326 @default.
• W2897974085 cites W2052606608 @default.
• W2897974085 cites W2053021047 @default.
• W2897974085 cites W2055642563 @default.
• W2897974085 cites W2056016033 @default.
• W2897974085 cites W2058968319 @default.
• W2897974085 cites W2060263803 @default.
• W2897974085 cites W2061545526 @default.
• W2897974085 cites W2063207603 @default.
• W2897974085 cites W2064953660 @default.
• W2897974085 cites W2067469839 @default.
• W2897974085 cites W2068013339 @default.
• W2897974085 cites W2070903666 @default.
• W2897974085 cites W2070999702 @default.
• W2897974085 cites W2071146637 @default.
• W2897974085 cites W2071816962 @default.
• W2897974085 cites W2072540192 @default.
• W2897974085 cites W2073440374 @default.
• W2897974085 cites W2073819646 @default.
• W2897974085 cites W2074857962 @default.
• W2897974085 cites W2076830797 @default.
• W2897974085 cites W2080545737 @default.
• W2897974085 cites W2082083680 @default.
• W2897974085 cites W2082282658 @default.
• W2897974085 cites W2084758579 @default.
• W2897974085 cites W2085069493 @default.
• W2897974085 cites W2085366528 @default.
• W2897974085 cites W2086885752 @default.
• W2897974085 cites W2087516645 @default.

• next