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- W2898041669 abstract "Copyright information: Taken from The extracellular matrix, p53 and estrogen compete to regulate cell-surface Fas/Apo-1 suicide receptor expression in proliferating embryonic cerebral cortical precursors, and reciprocally, Fas-ligand modifies estrogen control of cell-cycle proteinsBMC Neuroscience 2004;5():11-11.Published online 23 Mar 2004PMCID:PMC395829.Copyright © 2004 Cheema et al; licensee BioMed Central Ltd. This is an Open Access article: verbatim copying and redistribution of this article are permitted in all media for any purpose, provided this notice is preserved along with the article's original URL. , : Our data shows that during cell cycle, cell-surface Fas expression is highest in neuroblasts that also exhibit the highest level of BrdU incorporation. Such a relationship would occur at the end of S-phase, perhaps reflecting DNA replication errors. Therefore, Fas expression (indicated in the cartoon by a green peri-cellular halo), and hence suicide-sensitivity would be highest during the ventricular-fugal interkinetic movement of nuclei transitioning through G2. Resident Fas-ligand expressing cells (indicated by pacman figures) could eliminate defective Fas-expressing neuroblasts. , : Cortical neuroblasts utilize integrin-mediated signals to migrate along radial glia and into the laminae of the cortical plate []. Collagenase-A disrupts integrin-collagen interactions, and our data shows that collagenase-A leads to increased cell-surface Fas expression. Therefore, the induction of the Fas receptor may underlie the process of 'anoikis'. 'Anoikis' in turn, may protect the developing cerebral cortex from migration errors. Abbreviations: V = ventricular zone, VZ = ventricular zone, CP = cortical plate." @default.
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- W2898041669 date "2011-01-01" @default.
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- W2898041669 title "Models of Fas-mediated suicide sensitivity of precursors during cell cycle, and following disruption of cell-matrix interactions" @default.
- W2898041669 doi "https://doi.org/10.6084/m9.figshare.31199" @default.
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