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- W2898065401 endingPage "195" @default.
- W2898065401 startingPage "141" @default.
- W2898065401 abstract "The pulmonary endothelial cell forms a critical semi-permeable barrier between the vascular and interstitial space. As part of the blood-gas barrier in the lung, the endothelium plays a key role in normal physiologic function and pathologic disease. Changes in endothelial cell shape, defined by its plasma membrane, determine barrier integrity. A number of key cytoskeletal regulatory and effector proteins including non-muscle myosin light chain kinase, cortactin, and Arp 2/3 mediate actin rearrangements to form cortical and membrane associated structures in response to barrier enhancing stimuli. These actin formations support and interact with junctional complexes and exert forces to protrude the lipid membrane to and close gaps between individual cells. The current knowledge of these cytoskeletal processes and regulatory proteins are the subject of this review. In addition, we explore novel advancements in cellular imaging that are poised to shed light on the complex nature of pulmonary endothelial permeability." @default.
- W2898065401 created "2018-10-26" @default.
- W2898065401 creator A5012594718 @default.
- W2898065401 creator A5028860401 @default.
- W2898065401 creator A5039695549 @default.
- W2898065401 creator A5058607156 @default.
- W2898065401 date "2018-01-01" @default.
- W2898065401 modified "2023-09-30" @default.
- W2898065401 title "Cortical Actin Dynamics in Endothelial Permeability" @default.
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