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- W2898295655 abstract "Abstract Platelet signal transduction is the focus of this review. While ‘classic’ platelet signaling through G protein–coupled receptors in response to fluid-phase agonists has been extensively studied, signaling mechanisms linking platelet adhesion receptors such as GPIb-IX-V, GPVI and α2β1 to the activation of αIIbβ3 are less well established. Moreover, ‘non-haemostatic’ pathways can also activate platelets in various settings, including platelet–immune or platelet–tumour cell interactions, platelet responses to neutrophil extracellular traps, or stimulation by microbial pathogens. Genetically determined integrin variants can modulate platelet function and increase thrombogenicity. A typical example is the Pro33 (HPA-1b) variant of αIIbβ3. Recent advances in the genotype–phenotype relation of this prothrombotic variant and its impact on outside-in signaling will be reviewed." @default.
- W2898295655 created "2018-11-02" @default.
- W2898295655 creator A5031366610 @default.
- W2898295655 date "2018-11-01" @default.
- W2898295655 modified "2023-09-26" @default.
- W2898295655 title "Platelet Signaling in Primary Haemostasis and Arterial Thrombus Formation: Part 2" @default.
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- W2898295655 doi "https://doi.org/10.1055/s-0038-1675149" @default.
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