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- W2898366916 abstract "Introduction: Experimentally induced muscle damage (EIMD) is a common model in sports medicine, causing dysfunction as well as pain. Whereas primary dull pain is often referred to as delayed-onset muscle soreness (DOMS), the appearance of severe dysfunction and deep tissue pain refers to an increased trauma on a cellular and fascial level (Fleckenstein et al 2017). The sympathetic part of the autonomic nervous system has particularly been associated with the induction of neurogenic inflammation via vasodilation and plasma extravasation and the release of pro-inflammatory neuropeptides from its own nerve fibers (Fischer 2014; Gonzales et al 1989). Thus, we assume the sympathetic nerves to be a key mechanism mediating the late phase of EIMD." @default.
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- W2898366916 date "2018-10-01" @default.
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- W2898366916 title "The role of the autonomic nervous system in the development of exercise induced muscle damage: results of an RCT" @default.
- W2898366916 doi "https://doi.org/10.1016/j.jbmt.2018.09.062" @default.
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