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- W2898880854 abstract "Venous thromboembolism is a multifactorial disease. Major provoking factors are identified in 50–60 % of patients, but 40-50% of cases remains unexplained. In recent years, the role of chronic inflammatory disorders has been extensively investigated. Recently, immunothrombosis could explain some cases of “unprovoked” VTE. The inflammation may have an influence on three key elements of coagulation: initiation and propagation of coagulative cascade; downregulation of natural anticoagulants; inhibition of fibrinolitic pathway. Several components of the immune system, including cytokines, different types of leukocytes and platelets are involved in underlying inflammatory processes of VTE. In recent years, periodontal disease has been related to a large number of systemic disorders and relationship is observed between the presence of periodontitis and venous thromboembolic disease. Porphyromonas gingivalis, a Gram-negative anaerobic bacterium, is the major etiologic agent which contributes to chronic periodontitis. Periodonthopatogenic bacteria are able to provoke endothelial dysfunction, platelets activation, inflammatory cytokines production, and autoimmunity reactions thath lead to thrombin generation. The periodontitis could therefore explain a non-negligible percentage of unprovoked venous thromboembolic disease." @default.
- W2898880854 created "2018-11-09" @default.
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- W2898880854 date "2018-10-19" @default.
- W2898880854 modified "2023-10-17" @default.
- W2898880854 title "Periodontitis as A Risk Factor of Unprovoked Venous Thromboembolic Disease: An Emerging Association" @default.
- W2898880854 doi "https://doi.org/10.24087/iam.2018.2.12.694" @default.
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