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- W2899112623 abstract "The early stage of oncogenesis is linked to the disorder of the cell cycle. Abnormal gene expression often leads to cell cycle disorders, resulting in malignant transformation of human cells. Epstein–Barr virus (EBV) is associated with a diverse range of human neoplasms, such as malignant lymphoma, nasopharyngeal carcinoma and gastric cancer. EBV mainly infects human lymphocytes and oropharyngeal epithelial cells. EBV is latent in lymphocytes for a long period of time, is detached from the cytoplasm by circular DNA, and can integrate into the chromosome of cells. EBV expresses a variety of latent genes during latent infection. The interaction between EBV latent genes and oncogenes leads to host cell cycle disturbances, including the promotion of G1/S phase transition and inhibition of cell apoptosis, thereby promoting the development of EBV-associated neoplasms. Molecular mechanisms of EBV-driven cell cycle progression and oncogenesis involve diverse genes and signal pathways. Here, we review the molecular mechanisms of EBV-driven cell cycle progression and promoting oncogenesis." @default.
- W2899112623 created "2018-11-09" @default.
- W2899112623 creator A5007646350 @default.
- W2899112623 creator A5038292641 @default.
- W2899112623 creator A5048853031 @default.
- W2899112623 creator A5073446766 @default.
- W2899112623 date "2018-11-01" @default.
- W2899112623 modified "2023-10-17" @default.
- W2899112623 title "Molecular mechanisms of EBV-driven cell cycle progression and oncogenesis" @default.
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