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- W28993007 abstract "Shock is an acute diffuse reduction in effective tissue perfusion that leads to an imbalance of oxygen supply and demand, anaerobic metabolism, cellular dysfunction, metabolic disarray, and eventually death. Hemodynamic measurements are not as crucial to the definition or monitoring of shock as are oxygenation parameters. Abnormalities of intravascular volume, myocardial function, peripheral resistance, or distribution of blood flow account for the major pathophysiological problems of shock. Clinical classifications of shock include hypovolemic, cardiogenic, distributive (septic), and obstructive types. While the pathophysiology of each type of shock is different and complex, the final common pathway is poor perfusion, anaerobic metabolism, lactic acidosis, and mediator release with perpetual tissue damage. Shock is an evolving circulatory failure with variable physiologic patterns. Therapy should be directed toward the underlying pathophysiologic mechanisms and titrated to achieve optimal physiologic, oxygenation, and hemodynamic goals to maximize survival." @default.
- W28993007 created "2016-06-24" @default.
- W28993007 creator A5004360874 @default.
- W28993007 date "1990-06-01" @default.
- W28993007 modified "2023-10-18" @default.
- W28993007 title "Pathophysiology of Shock" @default.
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- W28993007 doi "https://doi.org/10.1016/s0899-5885(18)30816-5" @default.
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