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- W2899408056 endingPage "256" @default.
- W2899408056 startingPage "246" @default.
- W2899408056 abstract "Cardiac fibrosis, characterized by excessive accumulation of extracellular matrix, abolishes cardiac contractility, impairs cardiac function, and ultimately leads to heart failure. In recent years, significant evidence has emerged that supports the highly dynamic and responsive nature of the cardiac extracellular matrix. Although our knowledge of cardiac fibrosis has advanced tremendously over the past decade, there is still a lack of specific therapies owing to an incomplete understanding of the disease etiology and process. In this review, we attempt to highlight some of the recently investigated molecular determinants of ischemic and non-ischemic fibrotic remodeling of the myocardium that present as promising avenues for development of anti-fibrotic therapies." @default.
- W2899408056 created "2018-11-09" @default.
- W2899408056 creator A5014221538 @default.
- W2899408056 creator A5018496412 @default.
- W2899408056 creator A5053049885 @default.
- W2899408056 date "2019-04-01" @default.
- W2899408056 modified "2023-10-16" @default.
- W2899408056 title "Novel molecular therapeutic targets in cardiac fibrosis: a brief overview" @default.
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