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- W2899594937 abstract "Modern technologies relying on wireless communication systems have brought increasing levels of electromagnetic field (EMF) exposure. This increased research interest in the effects of these radiations on human health. There is compelling evidence that EMFs affect cell physiology by altering redox-related processes. Considering the importance of redox milieu in the biological competence of oocyte and sperm, we reviewed the existing literature regarding the effects of EMFs on reproductive systems. Given the role of mitochondria as the main source of reactive oxygen species (ROS), we focused on the hypothesis of a mitochondrial basis of EMF-induced reproductive toxicity. MEDLINE, Web of Science, and Scopus database were examined for peer-reviewed original articles by searching for the following keywords: “extremely low frequency electromagnetic fields (ELF-EMFs),” “radiofrequency (RF),” “microwaves,” “Wi-Fi,” “mobile phone,” “oxidative stress,” “mitochondria,” “fertility,” “sperm,” “testis,” “oocyte,” “ovarian follicle,” and “embryo.” These keywords were combined with other search phrases relevant to the topic. Although we reported contradictory data due to lack of uniformity in the experimental designs, a growing body of evidence suggests that EMF exposure during spermatogenesis induces increased ROS production associated with decreased ROS scavenging activity. Numerous studies revealed the detrimental effects of EMFs from mobile phones, laptops, and other electric devices on sperm quality and provide evidence for extensive electron leakage from the mitochondrial electron transport chain as the main cause of EMF damage. In female reproductive systems, the contribution of oxidative stress to EMF-induced damages and the evidence of mitochondrial origin of ROS overproduction are reported, as well. In conclusion, mitochondria seem to play an important role as source of ROS in both male and female reproductive systems under EMF exposure. Future and more standardized studies are required for a better understanding of molecular mechanisms underlying EMF potential challenge to our reproductive system in order to improve preventive strategies." @default.
- W2899594937 created "2018-11-16" @default.
- W2899594937 creator A5015854409 @default.
- W2899594937 creator A5024540410 @default.
- W2899594937 creator A5025086321 @default.
- W2899594937 creator A5027025954 @default.
- W2899594937 creator A5065071146 @default.
- W2899594937 creator A5076581545 @default.
- W2899594937 creator A5082165988 @default.
- W2899594937 date "2018-11-08" @default.
- W2899594937 modified "2023-10-11" @default.
- W2899594937 title "Role of Mitochondria in the Oxidative Stress Induced by Electromagnetic Fields: Focus on Reproductive Systems" @default.
- W2899594937 cites W1492783417 @default.
- W2899594937 cites W1533773296 @default.
- W2899594937 cites W1585469232 @default.
- W2899594937 cites W1594193345 @default.
- W2899594937 cites W1605224825 @default.
- W2899594937 cites W1669458375 @default.
- W2899594937 cites W1684983836 @default.
- W2899594937 cites W1723436886 @default.
- W2899594937 cites W181330873 @default.
- W2899594937 cites W1850322182 @default.
- W2899594937 cites W1897925090 @default.
- W2899594937 cites W1940273261 @default.
- W2899594937 cites W1966213621 @default.
- W2899594937 cites W1967487924 @default.
- W2899594937 cites W1971360460 @default.
- W2899594937 cites W1971757569 @default.
- W2899594937 cites W1971838539 @default.
- W2899594937 cites W1974584978 @default.
- W2899594937 cites W1976820865 @default.
- W2899594937 cites W1978357172 @default.
- W2899594937 cites W1981781541 @default.
- W2899594937 cites W1981865065 @default.
- W2899594937 cites W1982037401 @default.
- W2899594937 cites W1982130096 @default.
- W2899594937 cites W1988291781 @default.
- W2899594937 cites W1991815881 @default.
- W2899594937 cites W1992855205 @default.
- W2899594937 cites W1995811611 @default.
- W2899594937 cites W1996664822 @default.
- W2899594937 cites W1997677083 @default.
- W2899594937 cites W1999974146 @default.
- W2899594937 cites W2001707093 @default.
- W2899594937 cites W2005186665 @default.
- W2899594937 cites W2007185373 @default.
- W2899594937 cites W2009660996 @default.
- W2899594937 cites W2010915134 @default.
- W2899594937 cites W2011426940 @default.
- W2899594937 cites W2012611529 @default.
- W2899594937 cites W2018487102 @default.
- W2899594937 cites W2020679893 @default.
- W2899594937 cites W2024362879 @default.
- W2899594937 cites W2024485254 @default.
- W2899594937 cites W2025069184 @default.
- W2899594937 cites W2031199214 @default.
- W2899594937 cites W2031534477 @default.
- W2899594937 cites W2032743237 @default.
- W2899594937 cites W2033275550 @default.
- W2899594937 cites W2043326511 @default.
- W2899594937 cites W2044009682 @default.
- W2899594937 cites W2045111322 @default.
- W2899594937 cites W2047893062 @default.
- W2899594937 cites W2048170611 @default.
- W2899594937 cites W2050349140 @default.
- W2899594937 cites W2053788298 @default.
- W2899594937 cites W2055859834 @default.
- W2899594937 cites W2056421502 @default.
- W2899594937 cites W2056693269 @default.
- W2899594937 cites W2056922243 @default.
- W2899594937 cites W2057851932 @default.
- W2899594937 cites W2058209982 @default.
- W2899594937 cites W2064521716 @default.
- W2899594937 cites W2065648134 @default.
- W2899594937 cites W2065941658 @default.
- W2899594937 cites W2066492841 @default.
- W2899594937 cites W2069310726 @default.
- W2899594937 cites W2069708257 @default.
- W2899594937 cites W2070995184 @default.
- W2899594937 cites W2071997694 @default.
- W2899594937 cites W2072755387 @default.
- W2899594937 cites W2073308872 @default.
- W2899594937 cites W2075463312 @default.
- W2899594937 cites W2077359131 @default.
- W2899594937 cites W2077405447 @default.
- W2899594937 cites W2077486744 @default.
- W2899594937 cites W2080246545 @default.
- W2899594937 cites W2082569183 @default.
- W2899594937 cites W2082855606 @default.
- W2899594937 cites W2083113082 @default.
- W2899594937 cites W2089514662 @default.
- W2899594937 cites W2089999387 @default.
- W2899594937 cites W2090010005 @default.
- W2899594937 cites W2091290017 @default.
- W2899594937 cites W2094437995 @default.
- W2899594937 cites W2094463280 @default.
- W2899594937 cites W2097014482 @default.
- W2899594937 cites W2097310411 @default.