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- W2899876278 abstract "Introduction: Inward rectifier Kir2.1 channels primarily maintain resting membrane potential. Human cardiomyocytes and cardiac Purkinje fibers exhibit a similar phenomenon of the two levels of resting membrane potential in pathological hypokalemia, which is associated with cardiac arrhythmia. We recently demonstrated that Kir2.1 and K2P1 currents partially account for the phenomenon in human cardiomyocytes. However, the mechanism of how Kir2.1 channels similarly settle the two levels of resting membrane potential in various types of cardiac cells that may express different inward cation currents is not well understood. Hypothesis: we hypothesize that Kir2.1 channels settle two levels of resting membrane potential through a common mechanism by non-linear balances of various opposing cation currents. Methods: We employed conventional electrophysiological techniques to record membrane potentials and whole-cell currents in either CHO cells or human iPSC-derived cardiomyocytes that engineered to express indica..." @default.
- W2899876278 created "2018-11-16" @default.
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- W2899876278 date "2017-11-14" @default.
- W2899876278 modified "2023-09-28" @default.
- W2899876278 title "Abstract 14327: Kir2.1 Channels Settle Two Levels of Resting Membrane Potential With Inward Rectification" @default.
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