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• W2901813557 startingPage "39" @default.
• W2901813557 abstract "Cytosolic free calcium (Ca2+) is a second messenger regulating a wide variety of functions in blood cells, including adhesion, activation, proliferation and migration. Store-operated Ca2+ entry (SOCE), triggered by depletion of Ca2+ from the endoplasmic reticulum, provides a main mechanism of regulated Ca2+ influx in blood cells. SOCE is mediated and regulated by isoforms of the ion channel proteins ORAI and TRP, and the transmembrane Ca2+ sensors stromal interaction molecules (STIMs), respectively. This report provides an overview of the (patho)physiological importance of SOCE in blood cells implicated in thrombosis and thrombo-inflammation, i.e. platelets and immune cells. We also discuss the physiological consequences of dysregulated SOCE in platelets and immune cells and the potential of SOCE inhibition as a therapeutic option to prevent or treat arterial thrombosis as well as thrombo-inflammatory disease states such as ischemic stroke." @default.
• W2901813557 created "2018-11-29" @default.
• W2901813557 creator A5008888080 @default.
• W2901813557 creator A5014366123 @default.
• W2901813557 creator A5064493858 @default.
• W2901813557 creator A5068782139 @default.
• W2901813557 creator A5079584981 @default.
• W2901813557 date "2019-01-01" @default.
• W2901813557 modified "2023-09-29" @default.
• W2901813557 title "Store-operated calcium entry in thrombosis and thrombo-inflammation" @default.
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